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10 free AEMT practice questions: Pulmonary Disease Processes: Sorting the Dyspneic Patient

These are real questions from the same bank the app draws from. Each one is written to the NREMT AEMT content specifications and kept inside the AEMT scope of practice. Pick an answer and you get the full rationale, including why the other three options are wrong.

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Question 1 of 10

A 27-year-old patient with asthma is sitting forward on the edge of a chair after two nebulized bronchodilator treatments. Loud wheezing is heard across all lung fields and the patient answers questions one word at a time. P 124, R 30, BP 138/84, SpO₂ 91% on room air. Which finding tells the AEMT the most about how severe the patient's airway obstruction is?

Show the answer and rationale

Correct answer · Answering questions one word at a time

This patient is having a severe asthma attack, and the one word answers tell you how bad the obstruction is. Talking takes a steady stream of air pushed past the vocal cords. Count the words a patient gets out before stopping to breathe. That count tracks how much air is really moving through the narrowed lower airways. How the chest sounds does not tell you that. A patient answering one word at a time is barely moving air. One word answers point toward respiratory failure that is about to happen. Escalate now. Call for ALS, be ready to assist ventilations with a BVM, and transport without delay.

Why the others are wrong

Loud wheezing heard across all lung fields: Loud wheezing does tell you the lower airways are narrowed, so it sounds like a measure of how bad the attack is. Wheezing only happens when air moves fast enough through narrowed airways to make sound. A loud wheeze means air is still getting through. The loudness does not measure how blocked the airways are. A chest that goes silent is the one heading toward arrest.

A heart rate of 124 after two treatments: A heart rate of 124 belongs in your assessment, and it can come from hypoxia and the work of breathing. This patient has also had two nebulized bronchodilator treatments. A fast heart rate is an expected side effect of those drugs. The heart rate has two possible causes, so it cannot measure the airway by itself.

A respiratory rate of 30 breaths per minute: A respiratory rate of 30 is abnormal and fits a severe attack. The rate alone does not tell you how much air each breath moves. A patient taking 30 shallow breaths and a patient taking 30 effective breaths look the same on that number.

Question 2 of 10

A 74-year-old patient with a history of heart failure has worsening difficulty breathing. Fine crackles are heard at both bases, the neck veins are distended, and both ankles are swollen. The crew has laid the patient flat on the cot to carry them down the stairs, and the breathing became noticeably worse within seconds. How should the AEMT position this patient?

Show the answer and rationale

Correct answer · Upright with the legs hanging down

This patient has pulmonary edema, which is fluid backing up into the lungs from heart failure. The fine crackles at both bases, the distended neck veins, and the swollen ankles all fit heart failure. Sitting upright lets gravity pull some of the fluid away from the chest. The diaphragm also gets more room to work. Letting the legs hang down keeps blood pooled in the legs. Less blood then returns to a heart that already cannot keep up. That's why laying this patient flat made the breathing worse within seconds. Sit the patient back up, carry the patient down the stairs seated, and plan the carry around the position.

Why the others are wrong

Flat on the back with the head turned: Flat on the back is the position that just made this patient worse. Lying flat lets the blood pooled in the legs and abdomen drain back toward a heart that is already full. Turning the head does nothing about where the fluid goes.

On the left side with the knees drawn up: The left side with the knees drawn up is a comfort position for abdominal pain. This patient's problem is a heart overwhelmed with fluid, and lying on the side takes none of that load off.

Flat with the legs raised above the heart: Raising the legs above the heart sends more blood back to the center of the body. A patient in shock who needs more blood returning to the heart would get this position. This patient already has too much volume coming back, and raising the legs sends even more of it to the lungs.

Question 3 of 10

A nebulized bronchodilator works by relaxing the smooth muscle wrapped around the lower airways. Which patient's underlying problem will that action leave unchanged?

Show the answer and rationale

Correct answer · A patient whose alveoli are filled with fluid backed up from the heart

The bronchodilator leaves the patient with pulmonary edema unchanged. This patient's alveoli are filled with fluid backed up from the heart, as in left heart failure. The fluid is a pressure and volume problem, not a smooth muscle problem. There is no tight airway muscle for the bronchodilator to relax. Albuterol only acts on airway smooth muscle. Albuterol cannot pull fluid out of the alveoli or lower the pressure pushing fluid in. For this patient, CPAP comes before a nebulizer treatment. CPAP pushes fluid back into the blood vessels and takes some of the load off the heart.

Why the others are wrong

A patient whose lower airways are narrowed by bronchospasm: Bronchospasm is exactly what a nebulized bronchodilator treats. The smooth muscle is squeezing, and the drug relaxes the muscle. This patient's airway opens, and the question asks which patient's problem stays unchanged.

A patient whose airways are tightened by an allergic reaction: An allergic reaction has effects all over the body that albuterol will not fix. The airway tightening in an allergic reaction is still smooth muscle squeezing the lower airways. That muscle relaxes the same way it does in plain bronchospasm, so this patient's airway improves.

A patient wheezing during a chronic obstructive pulmonary disease flare: A COPD flare can look like the sickest patient in the group. The wheezing in a COPD flare comes from the same bronchospasm. How sick the patient is does not change what the drug acts on, so this airway still opens.

Question 4 of 10

An 86-year-old patient who took too much of a prescribed opioid is breathing without assistance while waveform capnography runs. Over ten minutes the EtCO₂ has climbed from 44 mmHg to 58 mmHg, and the waveform has held its normal rectangular shape the whole time. What does that pattern indicate?

Show the answer and rationale

Correct answer · Hypoventilation, since the shape held while the number rose

This patient is hypoventilating from the opioid. The EtCO₂ climbed from 44 to 58 over ten minutes, and the waveform kept its normal rectangular shape the whole time. Read the shape and the number as two separate pieces of information. A waveform that slants upward tells you the airways are narrowed. A normal rectangular shape tells you the air is getting out of the lungs fine. The climbing number tells you this patient is not moving enough air each minute, so carbon dioxide is building up. In an overdose, a rising number with a normal shape is your sign to start supporting this patient's ventilations.

Why the others are wrong

Extra carbon dioxide produced by a rising temperature: A rising temperature does make the body produce more carbon dioxide. When a patient is breathing enough, the extra carbon dioxide gets blown off. A temperature change alone does not push the number from 44 to 58 like this.

A leak in the sampling line diluting the sample: A leak in the sampling line is worth checking any time a number surprises you. A leak pulls room air into the sample. Room air moves the reading down, not up.

Bronchospasm narrowing the lower airways: Bronchospasm can push the EtCO₂ up in a patient who is getting tired, so the direction fits. Bronchospasm also bends the waveform into an upslope. This patient's waveform held its normal shape.

Question 5 of 10

A 58-year-old patient with difficulty breathing has a saturation of 85% on room air, so an AEMT applies an NRB at 10 L per minute. The reservoir bag empties completely every time the patient inhales. The vital signs are BP 148/88, P 112, R 26, and SpO₂ 88% on the mask. What should the AEMT do?

Show the answer and rationale

Correct answer · Raise the flow until the bag stays partly inflated through each breath

The NRB flow is too low for how hard this patient is breathing. The reservoir bag empties completely every time the patient inhales, and the SpO₂ is only 88% on the mask. The reservoir bag holds the oxygen this patient breathes in on every breath. The flow from the tank refills the bag between breaths. A bag that goes completely flat means this patient is breathing in more oxygen than 10 L per minute can refill. Once the bag is flat, the rest of that breath is room air leaking in around the mask. You do not need a different device here, only more flow. An NRB runs from 10 to 15 L per minute. Turn the flow up until the bag still has some oxygen left in it at the end of each breath.

Why the others are wrong

Leave the flow where it is, since a bag that empties shows the oxygen is being used: Some shrinking of the reservoir bag on each breath is normal. A bag that empties completely is a different finding. The flow ran out before the breath did, and this patient finished that breath on room air.

Switch to a simple face mask, which has no reservoir bag to empty: Changing devices makes sense when the one you have is failing. A simple face mask gives roughly 35 to 60 percent oxygen. That is less than the NRB was already giving this hypoxic patient.

Recheck the mask seal, since a leak at the cheeks is what empties the bag: A poor mask seal does lower the oxygen this patient gets, so checking the seal is never wasted. A leak at the cheeks lets room air in around the edges. The patient then pulls less from the bag, so the bag would empty more slowly, not faster. The seal is not what is emptying this bag.

Question 6 of 10

First responders placed an NRB at 15 L per minute on a 62-year-old patient about ten minutes ago. The AEMT arrives to find the patient unresponsive, breathing 6 times a minute, with chest movement that is barely visible. The vital signs are BP 128/76, P 96, R 6, and SpO₂ 96% on the mask. Which statement describes what that mask is doing for this patient?

Show the answer and rationale

Correct answer · It raises the oxygen in each breath, but it does not move air for the patient

This patient is getting oxygen but is not moving enough air. Oxygenation and ventilation are two different jobs. A patient can fail one while the other looks fine. The NRB is doing the oxygen job. The mask raises the oxygen in whatever air this patient pulls in, and that's why the SpO₂ reads 96%. The NRB cannot do the ventilation job. This patient is unresponsive and breathing 6 times a minute. The chest movement is barely visible. Breathing that slow and shallow does not move enough air to clear carbon dioxide. Turning the flow up does not fix the problem, because a mask has no way to push a breath in. The bag-valve mask is the only device you carry that moves air for this patient.

Why the others are wrong

It is doing both jobs, since oxygen at 15 L per minute pushes air into the chest: Fifteen liters a minute is the highest flow you run through a mask. A flow that high can seem like it is pushing air. The flow only fills the mask and the reservoir bag. The oxygen never becomes a breath, because the mask has no seal and nothing squeezing air into the chest.

It is clearing carbon dioxide, because the valve vents each exhalation out of the mask: The valve does let each exhalation leave the mask instead of going back into the reservoir bag. Getting carbon dioxide out of the mask is different from getting it out of the patient. Carbon dioxide only leaves the patient when enough air moves in and out of the lungs.

It is all this patient needs while the saturation stays at or above 94 percent: A saturation of 96% is a reassuring number, and it tells you the oxygen side is covered for now. The pulse oximeter only reports one of the two jobs. This patient is breathing 6 times a minute with almost no chest movement, and that is the ventilation job failing.

Question 7 of 10

A patient is wheezing, and the crew is deciding whether a nebulized bronchodilator is indicated. What decides that question?

Show the answer and rationale

Correct answer · The history around the wheeze, not the wheeze by itself

The history around the wheeze decides whether a bronchodilator is indicated. A patient with known asthma or COPD and a trigger points to bronchospasm. In bronchospasm, inflamed and tight smooth muscle narrows the airways. A beta-agonist nebulizer relaxes that muscle and opens the airways back up. A history of orthopnea, paroxysmal nocturnal dyspnea, jugular venous distension, and crackles in both lung bases points to pulmonary edema from heart failure. In pulmonary edema, fluid fills the alveoli and presses on the small airways. That fluid makes the same wheezing sound. A bronchodilator does nothing to move the fluid. Get the history before you decide on a nebulizer.

Why the others are wrong

Whether the saturation has fallen below 94% on room air: A saturation below 94% on room air is a reason to give oxygen, and a low saturation tells you the patient is in trouble. The saturation measures how well oxygen gets into the blood. The saturation does not tell you what is narrowing the airway, so it cannot tell you whether to nebulize.

Whether the wheeze is heard on inspiration or on expiration: A wheeze heard only on expiration versus on both inspiration and expiration does show how far the narrowing has progressed. A wheeze on both phases still does not tell you whether asthma or heart failure caused the narrowing. The nebulizer decision depends on that difference.

The loudness of the wheeze on auscultation: A louder wheeze sounds like it should mean worse obstruction. Loudness only reflects how much turbulent air is moving through the narrowed airway. A silent chest from near-total obstruction is more dangerous than a loud wheeze. Neither loudness nor silence tells you whether bronchospasm or fluid caused the narrowing.

Question 8 of 10

An 11-year-old patient in respiratory distress has been retracting hard for twenty minutes. Over the last two minutes the retractions have eased, the patient has gone quiet, and the overall appearance has worsened. The vital signs are BP 106/62, P 138, R 16, and SpO₂ 94% on oxygen. What does that change represent?

Show the answer and rationale

Correct answer · Exhaustion, and the point where ventilation gets supported

This child is exhausted, and this is the point where you support ventilation. The retractions have eased and the child has gone quiet, but the overall appearance has worsened. A child who stops working to breathe while looking worse is running out of energy, not getting better. In a child, you see respiratory failure in the appearance before the pulse oximeter shows it. A saturation of 94% on oxygen does not change what the appearance is telling you. The respiratory rate has also fallen to 16. Falling effort with a worsening appearance is when you start BVM ventilation.

Why the others are wrong

A response to the oxygen that should be given more time: Oxygen does help, and giving a treatment time to work is usually wise. Here the saturation of 94% is the number that lags behind. Waiting on that number delays ventilation this child already needs.

A normal pause between episodes of increased effort: Children do tire and rally in waves, so watching for a pattern is reasonable. A quiet child who looks worse, with a respiratory rate that has fallen to 16, is not resting between episodes.

Improvement, since the work of breathing has come down: Less work of breathing is the goal of everything you are doing for this child. In a child, effort dropping while the appearance gets worse is exhaustion, never improvement.

Question 9 of 10

A 22-month-old patient has had a barking cough and noisy breathing all evening, with loud stridor at rest on arrival. Ten minutes later the stridor is much softer, the patient is limp against the caregiver, and the lips have turned cyanotic. The vital signs are BP 88/52, P 172, R 48, and SpO₂ 89% on oxygen. What does the softer stridor mean?

Show the answer and rationale

Correct answer · Less air is moving, and the patient is getting worse

The softer stridor means less air is moving, and this child is getting worse. Stridor is the sound of air forcing its way past a narrowed upper airway. Making that sound takes airflow. Once the narrowing gets bad enough, the stridor fades because there is not enough air moving to make it. This child is also limp against the caregiver, with cyanotic lips and a saturation of 89% with oxygen already running. This child is heading toward respiratory arrest, not improving.

Why the others are wrong

The oxygen has taken effect and the sound will keep fading: Oxygen is the right thing to give, and oxygen does help this child. Oxygen does not shrink swelling in the upper airway. The saturation is still 89% with the oxygen running.

The swelling has come down and the patient is improving: Less noise would mean less swelling if the child also looked better. This child is limp and cyanotic. The stridor is quieter because airflow is being lost, not because the swelling went down.

The stridor has moved lower into the chest as a wheeze: Breath sounds can change as a child's condition changes. Stridor comes from a narrowed upper airway. Stridor does not move down into the chest and turn into a wheeze.

Question 10 of 10

An AEMT gave a nebulized bronchodilator to a 27-year-old patient with a history of asthma. Before the treatment, waveform capnography showed an expiratory segment that sloped upward without reaching a flat plateau, and the EtCO₂ was 52 mmHg. After the treatment, the expiratory segment rises steeply and holds a flat plateau, and the EtCO₂ is 43 mmHg. The patient now speaks in longer sentences and the accessory muscle use has decreased. How should the AEMT interpret this change?

Show the answer and rationale

Correct answer · Expiratory airflow has improved

The bronchodilator worked, and expiratory airflow has improved. Before the treatment, the waveform sloped upward and never reached a flat plateau. That sloped shape comes from the narrowed lower airways of asthma. Narrowed airways empty at different speeds, so exhaled gas keeps trickling to the sensor all the way through the breath out. After the treatment, the waveform rises steeply and holds a flat plateau. A flat plateau means the alveoli are emptying together again. The EtCO₂ dropped from 52 to 43 because trapped carbon dioxide is finally getting out. The patient also speaks in longer sentences and uses fewer accessory muscles, which matches the improved airflow.

Why the others are wrong

The sensor has become disconnected: A disconnected sensor makes the waveform suddenly disappear. This waveform still has a clear shape with a flat plateau and a reading of 43. Exhaled gas has to reach a connected sensor to make that waveform.

The patient is now hypoventilating: Hypoventilation makes the EtCO₂ go up. This EtCO₂ went down, from 52 to 43. The patient is also moving more air now, speaking in longer sentences with less accessory muscle use.

Air trapping has become more severe: Worsening air trapping makes the waveform slope even more and pushes the plateau further out of reach. This waveform went back toward a rectangle. Worse trapping would also push the EtCO₂ up, and this number came down.

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